TY  - JOUR 
AU  - Siguero-Álvarez, M.
AU  - Salguero-Jiménez, A.
AU  - Grego-Bessa, J.
AU  - de la Barrera, J.
AU  - MacGrogan, D.
AU  - Prados, B.
AU  - Sánchez-Sáez, F.
AU  - Piñeiro-Sabarís, R.
AU  - Felipe-Medina, N.
AU  - Torroja, C.
AU  - Gómez, M.J.
AU  - Sabater-Molina, M.
AU  - Escribá, R.
AU  - Richaud-Patin, I.
AU  - Iglesias-García, O.
AU  - Sbroggio, M.
AU  - Callejas, S.
AU  - O'Regan, D.P.
AU  - McGurk, K.A.
AU  - Dopazo, A.
AU  - Giovinazzo, G.
AU  - Ibañez, B.
AU  - Monserrat Iglesias, Lorenzo
AU  - Pérez-Pomares, J.M.
AU  - Sánchez-Cabo, F.
AU  - Pendas, A.M.
AU  - Raya, A.
AU  - Gimeno-Blanes, J.R.
AU  - de la Pompa, J.L.
PY  - 2023
SN  - 1524-4539
UR  - http://hdl.handle.net/20.500.11940/20980
AB  - BACKGROUND: The complex genetics underlying human cardiac disease is evidenced by its heterogenous manifestation, multigenic basis, and sporadic occurrence. These features have hampered disease modeling and mechanistic understanding. Here, we show...
LA  - eng
KW  - Humans
KW  - Animals
KW  - Mice
KW  - Bicuspid Aortic Valve Disease
KW  - Induced Pluripotent Stem Cells
KW  - Heart Defects, Congenital
KW  - Cardiomyopathies
KW  - Myocytes, Cardiac
KW  - Aortic Valve
KW  - Transcription Factors
KW  - Chromosomal Proteins, Non-Histone
TI  - A Human Hereditary Cardiomyopathy Shares a Genetic Substrate With Bicuspid Aortic Valve
DO  - 10.1161/circulationaha.121.058767
T2  - Circulation
M2  - 47
KW  - AS A Coruña
KW  - CHUAC
VL  - 147
ER  -