TY  - JOUR 
AU  - Herrer, I.
AU  - Roselló-Lletí, E.
AU  - Ortega, A.
AU  - Tarazón, E.
AU  - Molina-Navarro, M. M.
AU  - Triviño, J. C.
AU  - Martínez-Dolz, L.
AU  - Almenar, L.
AU  - Lago Paz, Francisca
AU  - Sánchez-Lázaro, I.
AU  - González Juanatey, José Ramón
AU  - Salvador, A.
AU  - Portolés, M.
PY  - 2015
SN  - 1755-8794
UR  - http://hdl.handle.net/20.500.11940/8240
AB  - BACKGROUND: Ischemic cardiomyopathy (ICM) is characterized by transcriptomic changes that alter cellular processes leading to decreased cardiac output. Because the molecular network of ICM is largely unknown, the aim of this study was to characterize...
LA  - eng
KW  - Adult
KW  - Aged
KW  - Antigens, Nuclear
KW  - Apoptosis
KW  - Autoantigens
KW  - B-Cell Lymphoma 3 Protein
KW  - CCAAT-Enhancer-Binding Protein-delta
KW  - Cardiomyopathies
KW  - Female
KW  - Gene Expression Profiling
KW  - Gene Expression Regulation
KW  - Humans
KW  - Male
KW  - Middle Aged
KW  - Myocardial Ischemia
KW  - Myocardium/pathology
KW  - Principal Component Analysis
KW  - Proto-Oncogene Proteins
KW  - Real-Time Polymerase Chain Reaction
KW  - Repressor Proteins
KW  - Sequence Analysis, RNA
KW  - Trans-Activators
KW  - Transcription Factors
KW  - Transcription, Genetic
KW  - Transcriptome
TI  - Gene expression network analysis reveals new transcriptional regulators as novel factors in human ischemic cardiomyopathy
DO  - 10.1186/s12920-015-0088-y
T2  - BMC Medical Genomics
VL  - 8
ER  -