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dc.contributor.authorPérez Encinas, Manuel Mateo 
dc.contributor.authorQuinteiro García, Celsa 
dc.contributor.authorSalas, Elisa M.
dc.contributor.authorGarcía-Barchino, María J.
dc.contributor.authorLabiano, Sara
dc.contributor.authorShugay, Mikhail
dc.contributor.authorGarcía-Delgado, Marina
dc.contributor.authorVizmanos, José L.
dc.contributor.authorNovo, Francisco J.
dc.date.accessioned2017-06-07T07:23:03Z
dc.date.available2017-06-07T07:23:03Z
dc.date.issued2011
dc.identifier.issn1947-6027
dc.identifier.urihttp://hdl.handle.net/20.500.11940/5812
dc.description.abstractA search for genes potentially regulated by STAT5 identified leukemia inhibitory factor (LIF) as a good candidate. Using various experimental approaches, we have validated LIF as a direct transcriptional target of STAT5 in myeloid cell lines: STAT5 binds to LIF promoter, and LIF expression is increased after activation of the JAK2/STAT5 pathway.We also found that LIF expression is significantly increased in patients with chronic myeloproliferative neoplasms with and without activating mutations of the pathway, indicating that LIF might play an important role in STAT5-mediated oncogenesis.en
dc.language.isoeng
dc.titleLIF, a novel STAT5-regulated gene, is aberrantly expressed in myeloproliferative neoplasms.
dc.typeArtigoes
dc.authorsophosSalas, Elisa M
dc.authorsophosGarcía-Barchino, María J
dc.authorsophosLabiano, Sara
dc.authorsophosShugay, Mikhail
dc.authorsophosPérez-Encinas, Manuel
dc.authorsophosQuinteiro, Celsa
dc.authorsophosGarcía-Delgado, Marina
dc.authorsophosVizmanos, José L
dc.authorsophosNovo, Francisco J;
dc.identifier.doi10.1177/1947601911420139
dc.identifier.pmid21901172
dc.identifier.sophos10248
dc.issue.number5
dc.organizationConsellería de Sanidade::Fundación pública Galega de Medicina Xenómica
dc.organizationServizo Galego de Saúde::Estrutura de Xestión Integrada (EOXI)::EOXI de Santiago - Complexo Hospitalario Universitario de Santiago::Hematoloxía clínica
dc.page.initial593
dc.page.final596
dc.rights.accessRightsopenAccess
dc.typesophosArtículo Original
dc.volume.number2


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